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The current evidence points to direct mitochondrial reprogramming (upregulation of fatty acid -oxidation, anti-apoptotic, and antioxidant pathways) triggered by GHRP-6 binding to GHS-R1a on cardiac myocytes a peripheral receptor population also present in hexarelin cardiac studies, but with a distinct binding site

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One prior report described PSD95-positive elements contacting intestinal enterochromaffin cells of the mouse 39 , but neither provided quantitative data nor proof of staining specificity

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A lesser extent of elimination occurs via bile into the feces