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Evidence indicates that an acetaminophen overdose can cause mitochondrial dysfunction, either by covalently binding to mitochondrial proteins or through alternative mechanisms [101]

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These findings highlight the complexity of MCT targeting and the potential for resistance mechanisms [354]

Investigation of cell-cycle controlling system has reported vitamin D participation in cell cycle arrest through controlling expression of different regulatory molecules such as HIF 1a , p53, MYC, Ras, MAPK, BRCA1, and GADD 45 [34]

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