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doi: 10.1007/s12640-019-9998-3

Even months after the initial infection, patients frequently exhibit significantly elevated fasting blood glucose levels, forcing the body to continuously store excess circulating glucose as visceral fat

BPC-157 complements these mechanisms through its activation of VEGFR2 and eNOS pathways that promote angiogenesis and improve endothelial function, with the peptide's nitric oxide generation supporting vasodilation and reducing thrombotic risk while its growth factor sensitization enhances vascular responses to ischemic conditions

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