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In apo E / mice fed an adenine-rich diet, chronic kidney disease and cardiac hypertrophy were observed alongside elevated circulating TMAO levels

doi:10.1093/sysbio/syw055

[2][3] Because of its significant effect on growth and development as well as causing associated medical problems and reduced quality of life, childhood-onset GHD has been treated with replacement therapy for more than 30 years

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More broadly, a growing body of preclinical evidence suggests that certain postbiotic preparations may attenuate weight gain, reduce adiposity, and stimulate thermogenesis, including brown adipose tissue signaling, particularly in HFD-induced rodent models (114)