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Recent research has demonstrated that the pathophysiology of IBD is largely influenced by T cell-mediated immune responses in the intestinal mucosa, specifically the imbalance in the dynamic equilibrium between Th17 and Treg cells, which is crucial for the onset and progression of IBD (179)

The lncRNA UCA1 promotes the progression of UC through the miR-331-3p/BRD4 molecular axis

Certain amino acids have been the focus of research, such as isoleucine restriction, which has been demonstrated to extend mouse life expectancy (Aman 2023)

Lack of effect of oral sulforaphane administration on Nrf2 expression in COPD: A randomized, double-blind, placebo controlled trial