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Finally, UCMSC-Exos were demonstrated to protect against burn-induced acute lung injury by inhibiting ferroptosis by activating the Nrf2/HO-1 pathway [213]

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761 The overexpression of CYP1B1 increases the ubiquitination and degradation of ACSL4, and melatonin could inhibit ferroptosis by regulating the ACSL4/CYP1B1 pathway, significantly reducing the cerebral ischemic area and neuron loss in MCAO mice

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INR649 Inclusive of all Taxes

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3 (May 3, 2016): 30812

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