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A satiated state was produced by administering nicotine throughout the study session with a patch, gum, or nasal spray, providing the benefit of acute nicotine exposure ( There are two primary theories proposed to explain the pro-cognitive effects of nicotine in SZ, and the relationship to the increase prevalence of smoking in SZ

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15 To further verify whether the increase in cholesterol levels induced by cigarette smoke in airway epithelial cells contributes to cigarette smoke-induced airway inflammation through ROS promotion, we treated HBE cells with water-soluble cholesterol and detected ROS levels in airway epithelial cells using flow cytometry (Figure 6A) and the secretion levels of inflammatory factors IL6 and IL8 using qPCR (Figure 6B)

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