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However, examination of the kinetics of observed responses revealed that while PF 06882961 had a similar profile to GLP-1 for induction of G protein conformational changes, calcium signalling and pERK1/2, it was slower at inducing -arrestin recruitment and internalisation ( Figure S1 )

Under physiological conditions, it supports synaptic plasticity [346], whereas under pathological states, such as during astrocytic necroptosis, proBDNF may exert neurotoxic effects and promote apoptotic signaling [349]