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Various types of environmental stress may trigger the production of these deleterious compounds, suggesting their protective role, e.g., under oxidative stress (Reverberi et al., 2010)

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The dominant categories are cell-culture studies in dermal fibroblasts and keratinocytes (collagen biochemistry, gene-expression observations, signal-transduction effects), copper-chemistry studies (chelation stability, antioxidant chemistry), and small-animal wound-repair models

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