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P grund av dess regenerativa egenskaper har det ocks visat sig stimulera proliferationen av senceller och kan vara anvndbart vid regenerering av neuronala celler

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Although the precise mechanisms of HSD17B13 variants in MASH remain unclear, studies have identified that protein kinase A-mediated phosphorylation of HSD17B13 interacts with adipose triglyceride lipase to promote lipolysis and suppress MASH 66

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